LYME DISEASE: SURVIVING TO FLUKES AND PARAGONIMUS!

This is not good news but very needed to open the eyes and properly treat this Lyme, because Borrelia enters the immune system some big bad opportunistic bugs show up and if not treated well, there is no recovery at all... many Lymies have shown to have FLUKES OR PARAGONIMUS... sometimes Praziquantel could help (I found it in Zimectrin Gold for horses and there is a human version in the US); but unfortunately the only medicine that really seems to work for treating flukes is  called TRICLABENDAZOLE, but the human version Fasinex is almost impossible to find anywhere in the world,  so most countries have to treat with the veterinary version.  If the person has flukes or paragonimus could get cancer if not properly treated and even after being treated; most lung problems are from the flukes, most liver problems are from these too and they also could cause intestinal parasitic infection.  Please note these super mega bugs are not being treated with the antibiotics you have been taking, nor the IV abx, nor oral and less herbals; if the person has flukes the medicine has to be as strong and equivalent as the bug.  
This is not bacteria, nor filarial parasites, nor the common spaghetti worms some might have seen in their toilets, these are the masters of disguise, real huge monster demons!  
 The way to test is in stool samples if the lab is looking for them, or if you see your "samples" like if you had eaten black beans... or with some brown seeds like whole almonds... plus the symptoms: Symptoms of parasite infection, specifically fascioliasis "include headaches, rashes, muscle pain, jaundice, abdominal pain, loss of appetite, anemia, nausea, and vomiting".  - And there might be other testing like DNA art testing or the biofeedback machines?  those might save our lives I think if we get a proper dgx.

This is other reading that states that the other anthelmintics could work but it is an older article, I present it here just for information: "Symptoms of schistosomiasis appear in three distinct phases.  In the initial phase, symptoms include:  fever, skin rash, abdominal pain, bronchitis, enlargement of the liver and spleen, and diarrhea.  In the intermediate phase, symptoms include pathological changes in the intestinal and urinary tracts, and eggs in the urine and feces.  The final phase results in complications involving major bodily systems."

"Diagnosis is accomplished by finding eggs in the urine or feces. The most effective drugs for treating schistosomoiasis are organic trivalent antimonials.  However, these drugs are toxic to humans, and of these drugs, only pentavalent stibogluconate is still in use.

The current drug of choice is praziquantel.  Other drugs used to treat schistosomiasis include metrifonate, oxamniquine, bithionol, albendazole, or mebendazole.  The mechanism of these modern drugs is not known.  However, it is known that one of the drugs (albendazole) starves the parasite and its offspring."... NOT TRUE, TRICLABENDAZOLE IS THE CHOICE NOW!

If I may add, I found a veterinary medicine called TRIVENTOR that contains all the anthelmintics such as Triclabendazole, febendazole, Ivermectina and Praziquantel - does not contain Albendazole but I guess it is because the Albenda is the one that most probably hurt the Central Nervous system, but opposite of what we think, Albenda is one of the antiparasitics preffered by the American Doctors because they do not know the other anthelmintics plus they ignore the studies about the central nervous system...

Anyways, I'm talking about monsters, huge, affecting Lyme patients who's bodies and immune system cannot combat by itself!  Hope we all could get tested and treated against FLUKES!.
Read this: 
"Symptoms of schistosomiasis are caused by the body's reaction to the eggs produced by worms, not by the worms themselves.
Digestive symptoms: The following list incomplete:
abdominal pain, nausea, vomiting, inability to pass gas, abdominal pain on breathing, constipation, diarrhea, abdominal sensitivity, abdominal pain near navel, abdominal pain on sneezing, abdominal pain on activity, right-side abdominal pain, abdominal pain on coughing, abdominal swelling, upper abdominal discomfort, abdominal burning, abdominal bloating, malabsorption, partial intestinal blockage, Diverticulosis* Acute kidney failure ... nausea, vomiting, diarrhea * Bowel Obstruction ... green vomit, constipation, fecal vomiting, vomiting, abdominal swelling http://www.earthtym.net/ref-fluke-info.htm"

The study made in Egipt that showed Triclabendazole as the proper medicine to treat for flukes here: 

http://www.emro.who.int/publications/emhj/0402/07.htm

Links of pictures of parasites a wonderful blog, by

Carol Poore, thank you:

http://www.epi-center.com/parasite.html

And more Links and info about triclabendazole,

http://www.drugs.com/mmx/triclabendazole.html

http://parasitology.com/worms/index.html

"[Fascioliasis (treatment)]¹—Triclabendazole is used as a primary agent in the treatment of fascioliasis caused by Fasciola hepatica (sheep liver fluke) and Fasciola gigantica (giant liver fluke). ^{{01} {02} {07} {08} {11} {13}}

[Paragonimiasis (treatment)]¹—Triclabendazole is used as an alternative agent in the treatment of paragonimiasis caused by Paragonimus westermani (lung fluke) ^{01}"
Mechanism of action/Effect:

Fasciolicidal not only against the adult worms present in the biliary ducts, but also against the immature larval stages of Fasciola migrating through the hepatic parenchyma; the mechanism of action is not thoroughly understood; however, triclabendazole is shown to penetrate into liver flukes by transtegumentary absorption followed by inhibition of the parasite's motility, probably related to the destruction of the microtubular structure, resulting in the death of the parasite; the immobilizing effect is paralleled by changes in the parasite's resting tegumental membrane potential, strongly inhibiting the release of proteolytic enzymes, a process that appears critical to the survival of the parasite ^{{05} {06} {07}}.
 Absorption:
Following oral administration, triclabendazole is absorbed from the gastrointestinal tract; absorption is increased twofold to threefold when triclabendazole is taken after a fatty meal ^{{02} {08}}. Distribution:
Triclabendazole and its metabolites attain high concentrations in the biliary tract, through which they are excreted back into the intestine over a period of several days ^{02}; less than 1% of orally administered triclabendazole is distributed into breast milk ^{09}. Biotransformation:
Triclabendazole is oxidized to sulfoxide (the primary metabolite) and sulfone (present in lesser amounts) over the first 24 hours following oral administration ^{02}.
 Time to peak concentration:
Approximately 8 hours ^{02}.
 Elimination:
    Fecal—Approximately 95% of orally administered triclabendazole (unchanged or as the primary metabolite) is excreted in the feces ^{09}.      Renal—Approximately 2% is excreted in the urine ^{09}. Precautions to Consider
Mutagenicity Studies conducted in laboratory animals have not shown triclabendazole to be mutagenic ^{{07} {08}}"

BUT THE ONLY POSSIBLE TRICLABENDAZOLE IS VETERINARY THE HUMAN VERSION IS STILL IN LIMBO LAND!   FASINEX IS IMPOSIBLE TO BUY, SO NADA, THERE IS NON HUMAN MED TO TREAT FOR FLUKES, NO EXISTE!
Only vet med and it is considered illegal in the US? ... :(
 

NOTE:  LYME THE ROLLERCOASTER BLOG DOES NOT OFFER,  DOESN'T GIVE,  NOR INTENDS TO GIVE ANY MEDICAL ADVISE NOR MEDICAL RECOMMENDATIONS. Just sharing my thoughts, research and experience!